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First, although GPER has been extensively studied in animal models (mice and rats), the phenotypes observed in these models may not fully replicate those observed in humans, thereby limiting the generalizability of the findings

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Transparency about peptide use is essential withholding this information can lead to misdiagnosis or inappropriate treatment

Mechanistically, p53 decreases the monoubiquitination rate of histone H2B on lysine 120 (H2Bub1) in the SLC7A11 gene regulatory region by facilitating the nuclear translocation of the deubiquitinase USP7, leading to the inactivation of SLC7A11 expression in erastin-treated cells [122]